What is the basic structure of a triglyceride?

What is the basic structure of a triglyceride?

Triglycerides are tri-esters consisting of a glycerol bound to three fatty acid molecules. Alcohols have a hydroxyl (HO–) group. Organic acids have a carboxyl (–COOH) group. Alcohols and organic acids join to form esters.

How would you describe a triglyceride?

Triglycerides are a type of fat (lipid) found in your blood. When you eat, your body converts any calories it doesn’t need to use right away into triglycerides. The triglycerides are stored in your fat cells. Later, hormones release triglycerides for energy between meals.

What are the properties of triglycerides?

They are non-polar, hydrophobic, insoluble in water and soluble in organic solvents. Specific gravity is less than water. Therefore fats and oil float on water.

What is the function of a triglyceride?

Triglycerides are lipids (waxy fats) that give your body energy. Your body makes triglycerides and also gets it from the foods you eat. High triglycerides combined with high cholesterol raise your risk of heart attack, strokes and pancreatitis.

What is an example of a triglyceride?

What are triglycerides? Triglycerides are fats from the food we eat that are carried in the blood. Most of the fats we eat, including butter, margarines, and oils, are in triglyceride form. Excess calories, alcohol or sugar in the body turn into triglycerides and are stored in fat cells throughout the body.

Does a triglyceride dissolve in water?

Triglycerides are completely insoluble in water.

What causes low steady state triglycerides in the liver?

Under physiological conditions, the low steady-state triglyceride concentrations in the liver are attributable to a precise balance between acquisition by uptake of non-esterified fatty acids from the plasma and by de novo lipogenesis, versus triglyceride disposal by fatty acid oxidation and by the secretion of triglyceride-rich lipoproteins.

How is hepatic triglyceride homeostasis achieved under normal conditions?

This review discusses the molecular mechanisms by which hepatic triglyceride homeostasis is achieved under normal conditions, as well as the metabolic alterations that occur in the setting of insulin resistance and contribute to the pathogenesis of NAFLD. Fatty Acids / metabolism Fatty Liver / metabolism*

Why is hepatic lipid accumulation not offset by fatty acid oxidation?

Increased hepatic lipid accumulation is not offset by fatty acid oxidation or by increased secretion rates of triglyceride-rich lipoproteins.