Is hemochromatosis classed as a disability UK?

Is hemochromatosis classed as a disability UK?

Is hemochromatosis classed as a disability UK?

Genetic haemochromatosis qualifies as a disability under the Equality Act 2010. Under the Act, genetic haemochromatosis represents a protected characteristic – a “physical or mental impairment” which has “a substantial and long-term adverse effect” on someone’s “ability to carry out normal day-to-day activities”.

What are some symptoms of hereditary hemochromatosis?

How do you know if you have hereditary hemochromatosis?

  • Feeling of tiredness or weakness,
  • Weight loss,
  • Joint pain,
  • Bronze or grey skin color,
  • Abdominal pain, and.
  • Loss of sex drive.

How does hemochromatosis make you feel?

You may feel a lack of energy, general weakness, and difficulty concentrating (“memory fog”). Women are more likely than men to report fatigue as an early symptom of hemochromatosis. Fatigue can be a symptom of complications of hemochromatosis, such as heart failure, cirrhosis of the liver, or diabetes.

Does hemochromatosis affect your teeth?

Genetic haemochromatosis (GH) is responsible for iron overload. Increased transferrin saturation (TSAT) has been associated with severe periodontitis, which is a chronic inflammatory disease affecting tissues surrounding the teeth and is related to dysbiosis of the subgingival microbiota.

What happens if hemochromatosis is not treated?

Untreated, hereditary hemochromatosis can lead to a number of complications, especially in your joints and in organs where excess iron tends to be stored — your liver, pancreas and heart. Complications can include: Liver problems. Cirrhosis — permanent scarring of the liver — is just one of the problems that may occur.

What happens if Hemochromatosis is not treated?

Does hemochromatosis affect sleep?

Many patients also have periodic limb movements in sleep (PLMS), and they may complain of insomnia and/or hypersomnia. Hereditary haemochromatosis is an autosomal recessive disease of iron metabolism in which increased intestinal absorption of iron leads to iron deposition in multiple organs.